This article details the science of how Methamphetamine (METH) addiction has been proven to cause serious fungal infections in users. These fungal infections that addicts develop that I contend are the main reason they engage in socially and morally unacceptable behavior and develop mental illness, which culminates into suicidal tendencies and nihilistic world views.
Often, just in a few short years, they go from looking like normal people to what we can call a demon-like appearance and behaviors. Hence, it makes them Meth Demons who seem to be hell-bent on destroying themselves and everyone around them.
My theory is that it is not just the METH that makes addicts crazy, but it is the fungi/molds that grow within their lungs and gastrointestinal tracts, which has been proven to be our second brain. Various studies have shown how quickly this weakens their immune system from ingesting a drug containing things like battery acid, fuels, antifreeze, and cold medicines combined with horrible eating habits, a severe deficiency of vitamins, a lack of sun, and a lack of sleep.
When you combine all these toxic ingredients with terrible health habits into a daily routine, studies show that METH addicts quickly develop serious fungal infections, and I believe it is these organisms that have turned parasitic within addicts that are manipulating them and controlling their actions via this second brain.
Please think about this.
In my experience, these addicts are humans. Many are good people who may be our sons and daughters who have ignorantly partaken in a deadly drug that can cause a severe medical condition that can destroy their health and steal their personalities and ultimately ruin their lives.
Who would tolerate and still use a drug like METH that destroys how we look, our relationships, how we think, feel, and act and eventually lead to our death?
Are the people who use these drugs still “in control of their bodies and minds” or is their drug use resulting in a “pathogen” that is hell-bent on mind-controlling their victims?
Just like they have done to other animals and insects throughout the world as I describe in my articles, “Lord of the Flies: Fungi controls fly’s mind as it preys and infects other flies,” and “Parasitic mold that controls cicadas mind and forces them to infect other insects.”
Before I began sharing my research, I would like you to think about some questions that I believe will give you context to my theory as you read further;
* Did you know that chronic METH use severely increases the chances of a systemic fungal infection in the lungs and brains of users?
* Did you know that both METH and molds/fungi cause biochemical, behavioral, and physiological abnormalities and psychosis?
* Did you know that both METH and molds/fungi can lead to long-term deterioration of attention, memory, and judgment?
* Did you know that both METH addicts and molds/fungi love sugar?
HOW METHAMPHETAMINE USE CAUSES SYSTEMIC FUNGAL INFECTIONS
In the United States of America, Methamphetamine (METH) addiction is one of the worst threats to our society because it adversely changes people’s behavior, making them more prone to crime and carriers of and transporters of various infectious diseases. One of these diseases that METH addicts are highly susceptible to developing and influencing their pathogenic behavior that I would like to bring to your attention is a systemic fungal infection.
It is essential to understand that METH use destroys your immune system and make addicts much more susceptible to infection. Several studies have been done over the last decade, showing the severe impact of methamphetamine on infection and immunity. A 2015 study had shown that as a result of drug use, our bodies create chemical defenses, which increases the pro-inflammatory responses, and the induction of oxidative stress pathways.
This causes significant neurotoxicities to arise, increasing the risk for acquiring transmissible microbes and other opportunistic infections such as systemic fungal infections; this research has been documented worldwide (Plankey et al., 2007; Volkow et al., 2007; Ye et al., 2008; Sutcliffe et al., 2009; Parry et al., 2011; Borders et al., 2013; Eugenin et al., 2013; Heninger and Collins, 2013; Khan et al., 2013; Stahlman et al., 2013; Liao et al., 2014).
According to the researchers in the study;
METH abrogates normal macrophage function, resulting in accelerated disease in murine histoplasmosis (Martinez et al., 2009). METH decreases phagocytosis and killing of H. capsulatum by primary macrophages. METH exposed H. capsulatum-infected mice have increased fungal burdens, increased pulmonary inflammation, and reduced survival.
METH exposure results in cytokine dysregulation, aberrant processing of yeasts within macrophages, and immobilization of MAC-1 receptors on the macrophage surface. Additionally, METH inhibits T cell proliferation and alters antibody production, both important components of adaptive immunity. Hence, it is established that METH alters the immune system of a mammalian host, resulting in enhanced disease (Martinez et al., 2009). (1)
It has been proven by science through various studies that METH has diverse effects on a person’s immunity, and it also stimulates fungal adhesion and biofilm formation in the lungs, which causes dissemination of the fungus from the respiratory tract into the brain. Meaning it provides the perfect environment in your body for molds/fungi to grow, reproduce, and become permanent residents in our lungs and brain.
This is when I believe the fungi become pathogenic and can seriously manipulate an addict’s mind to do its bidding like they have been proven to do in other insects.
It turns humans into walking and talking fungal parasites – AKA Demons!
To document these transformations in their appearance and in hopes to scare other people from using this demon drug, the Faces of Meth campaign was launched in 2004. Their goal was to show before and after pictures of users side by side, which proves the devastation that meth use causes to addicts – sometimes over the course of a few months.
According to a study done in 2013, researchers discovered that METH use has profound implications on tissue homeostasis and the host’s capacity to respond to invading pathogens such as Cryptococcus neoformans (C. neoformans). (2) Meaning, that people who use METH are weakened to the point that they do not have a healthy immune system which allows pathogens such as the microorganism known as fungi or molds in English that causes, or can cause, disease and damage in its host.
The researchers call this an “enhanced fungal invasion.”
According to the lead researcher, Luis Martinez of Long Island University-Post, in Brookville, New York and of Albert Einstein College of Medicine in The Bronx;
Martinez says this greater ability to cause disease in the lung may be due in part to simple electrical attraction.
Their analysis shows that METH imparts a greater negative charge on the surface of the fungal cells, possibly lending them a greater attraction to the surface of the lung and an enhanced ability to form a biofilm that can protect its members from attack by the immune system. The fungus also releases more of its capsular polysaccharide in METH-treated mice, which can help the organism colonize and persist in the lung.
He commented, “When the organism senses the drug, it basically modifies the polysaccharide in the capsule. This might be an explanation for the pathogenicity of the organism in the presence of the drug, but it also tells you how the organism senses the environment and that it will modify the way that it causes disease.”
But the fungus doesn’t stop in the lungs. “The drug stimulates colonization and biofilm formation in the lungs of these animals,” says Martinez. “And this will follow to dissemination to the central nervous system by the fungus,” Martinez says.
The conclusion of the study stated;
“METH promotes C. neoformans colonization of the lungs upon infection and subsequent biofilm formation. Our findings suggest that C. neoformans biofilms may act as a fungal reservoir, shielding single cells from phagocytic cells, which can later disseminate, especially to the CNS. Moreover, the drug causes profound defects in the integrity of the Blood-Brain-Barrier BBB in vivo, increasing permeability, and facilitating the transmigration of C. neoformans to the CNS.
METH-induced alterations to the molecules responsible for maintaining the integrity of the BBB provide an explanation for the susceptibility of a METH abuser to brain infection by HIV and other pathogens. Broadly, METH has diverse and pronounced detrimental effects on host immunity that can also enhance pathogen persistence and proliferation.”
I would like to point out that METH is a drug that acts upon the central nervous system and chronic meth abuse causes detrimental effects on host immunity, which can lead to the fungi/molds proliferating the lungs and the blood brain barrier through the central nervous system.
It is akin to the fungi taking a bullet train to your brain via the central nervous system.
In some people, the parasite-host manipulation from the GI Tract can start happening immediately or within days/weeks causing them to develop psychosis or go insane i.e.: parasite-controlled humans.
METH also causes the massive release of the neurotransmitters like dopamine, norepinephrine, and serotonin, and blocks their reuptake, leading to long-term deterioration of attention, memory, and judgment. (Downes and Whyte, 2005; Collins et al., 2014). That may be one reason why users make such poor choices repeatedly because they have forgotten how to be human, and the moral codes that govern our societies are swapped by the moral codes of the fungal parasite, of which there seem to be none.
Hence, you will know them by their fruits or, more appropriately, their moldy and decaying fruits.
Along with neuropsychiatric deficits, methamphetamine abusers suffer from mental illnesses such as anxiety, depression, and psychosis being the most commonly reported.
Now, let me turn your attention to the fact that the toxins produced by some fungi/molds are neurotoxins that are poisonous or destructive to brain and nerve tissue, which causes a condition known as neurotoxicity. The term neurotoxicity refers to damage to the brain or peripheral nervous system caused by exposure to toxins and myconeurotoxicity when a person is exposed to mold toxins, which I believe is exactly happening.
When mold mycotoxins cause neurotoxicity, it is called myconeurotoxicity, which refers to any adverse effects of exposure to mycotoxins or byproducts of primary and secondary mold metabolism, including volatile organic compounds (VOCs) on the structural or functional integrity of the developing or adult nervous system. Neuromycotoxic effects may involve a spectrum of biochemical, morphological, behavioral, and physiological abnormalities whose onset can vary from immediate to delayed actions, following exposure to mycotoxins. The duration of effects may be transient or persistent and result in disability in some individuals, while some may have life-threatening consequences. (3)
A November 2015 study of mice titled “Mold inhalation, brain inflammation, and behavioral dysfunction” was developed by researchers to show a mouse model to determine how mold exposure can lead to neurobehavioral dysfunction. The researchers had formed a hypothesis that mold inhalation, like a bacterial infection, activates an innate immune response triggering microglial activation with resultant behavioral dysfunction.
Here is an excerpt from the study:
“Deficits in contextual memory were correlated with numbers of amoeboid microglia and microglial size in the dorsomedial dentate gyrus. Spore inhalation increased the numbers of cells in the hippocampus expressing the proinflammatory cytokine interleukin-1beta (IL-1beta). Increased numbers of cells expressing IL-1beta in hippocampal CA1 were positively correlated with spatial memory deficits and increased fear.
Mold exposure also affected two of the three stages of neurogenesis. Inhalation of EX spores decreased numbers of immature new neurons in the dorsomedial hippocampus expressing doublecortin, while IN treatment decreased numbers of adult-born BrdU-labeled neurons that matured and expressed NeuN. Our data suggest that respiratory exposure to any mold, not just the particularly toxic ones like Stachybotrys, may be capable of causing brain inflammation, cognitive deficits, and emotional problems.” (4)
Immune system disorders and abnormal natural killer cell (NKC) activity was found in patients with chronic toxigenic mold exposure in a 2003 study. The major symptoms reported were headache, general debilitating pains, nose bleeding, fevers with body temperatures up to 40 degrees C (104 degrees F), cough, memory loss, depression, mood swings, sleep disturbances, anxiety, chronic fatigue, vertigo/dizziness, and in some cases, seizures.
The researchers found that the patient’s sleep could be disturbed by mycotoxins and exerted some rigorous effects on the circadian rhythmic processes resulting in sleep deprivation. Depression, psychological stress, tissue injuries, malignancies, carcinogenesis, chronic fatigue syndrome, and experimental allergic encephalomyelitis could be induced at very low physiological concentrations by mycotoxin-induced NKC activity.
The researchers concluded that chronic exposures to toxigenic mold could lead to abnormal NKC activity with a wide range of neurological consequences, some of which were headache, general debilitating pains, fever, cough, memory loss, depression, mood swings, sleep disturbances, anxiety, chronic fatigue, and seizures.
This research correlates with other studies that have focused on mold exposure, brain changes, and neuropsychological problems such as mild traumatic brain injury, dysregulation of emotions, decreased cognitive functioning, short-term memory loss, executive function/judgment, concentration, and hand/eye coordination.
It is important that you understand my theory that it is not the methamphetamine that is causing the mental illness and psychosis, but that it weakens the users’ immune system, leading to a systemic fungal infection, additional infections. For example, it is not just the fungus, Cryptococcus neoformans (C. neoformans) that people need to worry about but other fungi can cause infection, illness, disease, and death in addicts.
METH CAUSES CANDIDA INFECTIONS AND CANDIDA INFECTION CAUSE MENTAL ILLNESS, ITCHING SYSTEMS, AND MEMORY LOSS
Candidemia, a bloodstream infection caused by Candida species and is prevalent amongst IV Drug users of METH. Candidemia is typically considered a health care-associated infection, but injection drug use (IDU) has emerged as an increasingly common condition related to candidemia. Among 203 candidemia cases in the Denver metropolitan area during May 2017–September 2018, 11% of the cases were IV drug users and of which 73% reported using METH, according to research published by the CDC. (5)
Studies have shown that METH facilitates intracellular replication and inhibits intracellular killing of Candida albicans and Cryptococcus neoformans. (6)
Candida is a yeast-like fungus naturally found in small amounts in human digestive tracts, but drugs enhance its overgrowth like METH with its chronic use, poor diet, excess sugar intake, and lack of sleep. Users often report itching sensations and scratch their skin, creating sores sometimes all over their bodies and faces. Users have even made claims of bugs, worms, and or flies crawling underneath their skin.
What is interesting is that Candida infections cause burning, itching symptoms, thrush (rashes in the throat or mouth), and sexually transmittable genital yeast infections in men and women.
Candida infections are linked to mental illness and are more common among those with memory loss.
The same mental issues that chronic Meth users suffer from.
For example, in a 2016 study published in Science Daily, both men and women with schizophrenia or bipolar disorder who tested positive for Candida performed worse on a standard memory test than people with the same disorders who had no evidence of past infection. According to the lead researcher, Emily Severance, Ph.D., assistant professor of pediatrics and member of the Stanley Division of Developmental Neurovirology at the Johns Hopkins University School of Medicine;
“Although we cannot demonstrate a direct link between Candida infection and physiological brain processes, our data show that some factor associated with Candida infection, and possibly the organism itself, plays a role in affecting the memory of women with schizophrenia and bipolar disorder, and this is an avenue that needs to be further explored,” says Severance.
“Because Candida is a natural component of the human body microbiome, yeast overgrowth or infection in the digestive tract, for example, may disrupt the gut-brain axis.
This disruption, in conjunction with an abnormally functioning immune system, could collectively disturb those brain processes that are important for memory.”
“However, most Candida infections can be treated in their early stages, and clinicians should make it a point to look out for these infections in their patients with mental illness.” She adds that Candida infections can also be prevented by decreased sugar intake and other dietary modifications, avoidance of unnecessary antibiotics, and improvement of hygiene. (7)